Noticing very chunky young kids
![[Post New]](/jforum/templates/default/images/icon_minipost_new.gif){kind=icon}
Anonymous
So you didn't have a beagle or other breed known for overeating. Dog breeds vary, some will self-limit and others won't. Just like people. |
Anonymous
Hopefully by then you will have taught them to listen to their own hunger cues. Which is part of our jobs as parents. |
Anonymous
No it isn’t. Can we just stop this? It is a cultural shift to eat crap as the majority of your food intake, especially for low income. |
Anonymous
I’m sorry, I guess I must be dense. Can you break it down for me further, please? What exactly is complicated about the concept of two entirely different people having two entirely different caloric needs? |
Anonymous
Haven’t you been reading thisnthread? It’s very clear that all the little UMC Larlo’s and Larla’s are getting fat because of their extra servings of chicken thighs and soup 
|
Anonymous
Er, no. I’ve never still been hungry after two full servings of a complete dinner meal. And thankfully neither have my kids. This kid is missing his fullness cues somehow, which is why he is gaining weight. |
Anonymous
Lolz |
Anonymous
I think it is both. Many people eat a lot of crap food, and many people overeat in general. But wherever. All of your kids are likely to be fat eventually anyhow. It’s projected by 2050 >90% of adults will be overweight |
Anonymous
I think it's hard for people to feel they are denying their kids food- but I have seen that statistic too (or something very close to it). Parents have to get it in their heads that eating disorders aren't going to be the battle of most of the future generation. For the vast majority of people , it's going to be obesity. People need to parent accordingly. Nobody else is going to solve this problem for your kids. |
Anonymous
Dense isn't the word I would have chosen, but your moral certitude could use a little examination. Here's a little tidbit, with the full article linked below. "In the 2010s, as family doctors and school nurses across the country were instructing larger children to “eat less and exercise more,” clinicians and scientists who specialized in obesity were beginning to understand the phenomenon in a different way. They knew from seeing kids in the clinic that some of them couldn’t lose weight or maintain weight loss no matter how hard they tried. As far back as the early aughts, university hospitals had been performing bariatric surgeries on a small number of these children who fell into an obesity category the Centers for Disease Control and Prevention labels “severe.” It was, to a large degree, the study of post-bariatric patients that led doctors to see obesity not as a matter of simple arithmetic but as a “pathophysiological disorder” of the signals among a body’s gut, organs, hormones, fat tissue, and brain. Not all bariatric patients were able to maintain a lower weight, but for those who did, it seemed the operation had reduced their appetite — not only by limiting how much they could eat but also by rewiring their internal system. This new understanding, coupled with a goal of dispelling the belief that an inability to lose weight reflects a failure of will, led the American Medical Association to establish obesity as a disease in 2013. But insight into obesity’s internal mechanisms did little to shed light on its prevalence. Why were there so many more kids with obesity? And why were so many of them so much more obese? In 2008, 36.5 percent of children ages 2 to 19 were overweight or obese; by 2018, that percentage was 41.5. Prevalence was climbing in children of every racial group, with the highest growth among Black, Hispanic, and Native American communities. By the end of 2020, children who were already obese were gaining weight at an accelerated rate. Pediatricians use growth charts to measure how a child’s size compares with the norm, and as the obesity epidemic escalated, they found themselves plotting more and more patients at the chart’s upper reaches, often above the 95th percentile of BMI, and sometimes off the grid entirely. When a phenomenon becomes so widespread, scientists look for causes beyond individuals and to their environment. The increase in obesity obviously correlated with the food kids ate — addictive, palatable, accessible at all hours — and their lack of physical activity: the phones, yes, but also reduced recess hours, unsafe neighborhoods, and lockdowns. Poverty and hunger correlate with obesity, as do other traumatic childhood experiences such as the death of a parent or sexual abuse. The frontier of obesity research lies here, in understanding how these external factors, as pervasive and variable as the weather, interact with each individual body — disrupting the metabolism, activating genes, or miscuing hunger signals — to produce the condition physicians call “obesity.” A propensity for obesity is encoded in certain people’s genes. More than 70 genes correlated with obesity are already known, though the presence or absence of an obesity gene does not forecast a child’s future body shape. Nevertheless, the biggest known predictor of whether a child will develop obesity is if her parents have it; there is a 40 percent likelihood with one parent and an 80 percent likelihood with two. The question is which environmental factors (and in which combinations and at what levels of intensity) turn the genes “on.” Then, further into the realm of unknowns: What are the triggers for obesity beyond known genetic predispositions? Toxins in food may modify our DNA, an epigenetic disruption that can be passed to the next generation. Changes in an individual’s gut bacteria, caused by the biochemical composition of food, may have the downstream effect of altering metabolism. “There are many, many, many associations” between the environment and the body that can produce obesity, says Sarah Armstrong, a professor of pediatrics and an obesity specialist at Duke, “but no one smoking gun.” https://www.thecut.com/article/weight-loss-drugs-ozempic-kids-childhood-obesity.html |
Anonymous
That will certainly not happen with all of the highly effective weight loss medications currently on the market an in the development pipeline. Obesity will become a disease that is almost nonexistent among people that can afford these medications. |
Anonymous
And those people will have other health issues as the story will unfold |
Anonymous
Ingredients can be inexpensive. But there is time involved in making it into food. That is something the parents may lack. Also the possibility of living in food deserts. I remember my parents making a 45 min trip on the one day they both had off (one car only) so they could go to the nearest grocery store to get ingredients for food. |
Anonymous
There are thousands of genes associated with being overweight and obese. National health systems in many countries that have conducted large studies including 100k-1M+ people to find causal variants for obesity/weight gain. Of course it’s is easier for some people to stay thin than others, but environmental factors and personal choices are very important. In the 1950s, only 10% of US adults were obese. Now around 40% of US adults are obese. Diets have become terrible, portion sizes have become much larger, people are getting less physical activity. Genes explain some variation between people in terms of susceptibility to weight gain, but they explain almost none of the exponential growth in obesity over the last 70 years. Population level genetic susceptibility to diseases doesn’t change much in two generations. |
Anonymous
I don’t know anyone personally from Mexico that I can think of. I have family members and friends from Puerto Rico, Peru, Colombia, Argentina, Brazil, but no Mexican friends. |